The gene isn't uniformly distributed throughout the entire human race, some families have it, some don't.

My point was merely that this gene would've increased the likelihood of certain people living longer than others (without the gene) due to Vitamin D's *other* longevity-enhancing properties (e.g., resistance to cancer, increase in muscle/bone strength, etc.).

Some people are claiming that Vitamin D helps prevent colds, too, and it's the fact that people bundle-up in wintertime that tends to allow more colds to happen (while hikers expend enough energy that they tend to wear less while hiking, and therefore don't catch colds as often even though they're exposed to more "cold weather").

There's a study (published here on PhysOrg) that says that elderly women in nursing homes given higher than normal Vitamin D supplements not only have fewer fractures when they fall, but more interestingly, that they have fewer *falls* than the control group.

Researchers in Britain are suggesting that the Recommended Daily Allowance for Vitamin D should be increased to something in the 2000IU/day range, about 5x the current US RDA.

I think one of the other sources of Vitamin D (other than supplements) is fish, so maybe the populations that eat a lot of fish tend to live longer anyway from their diet. Other populations tend to get more Vitamin D from the sun, from doing a lot of work outside.

I'm not married to this theory, it just occurred to me, having heard a bald stand-up comic comment on how you never see a bald homeless guy (which is something of an exaggeration, but seems to have a grain of truth to the observation). I started going bald early, when I was in college. I spent most of the day inside, studying (and drinking). Maybe a coincidence, maybe a connection, I don't know. Just passing the idea along.

I'm not bald however i have the Vampire v hairline look that suggests a lot of testosterone.

Male-pattern baldness also seems to be associated with a lot of chest hair, which is also associated with high levels of testosterone.

I think Androgenic Alopecia probably evolved through sexual selection as an enhanced signal of aging and social maturity. Bald men are perceived as being older than men with a full head of hair. AGA is also very common among other primates, and is often used to convey increased status and maturity. Gorillas evolved anatomically enlarged forheads for this reason. A balding scalp enhances the apparent size of the forhead, and increases the area of face to display. Chimpanzees, orangutans, stump-tailed macaques and bald ukari monkeys all display baldness with age. The bald ukari uses an increased area of the face to display its bright red skin which is a sign of good health. Before our current accent on youth, AGA was probably an idicator of healthy genes as it usually occurs after sexual maturity at a later stage in life, and as our ancestry primates had shorter life-spans, surviving to the stage AGA occurs would of been a sign of health and longevity. Premature AGA probably evolved to give the appearence of being older and more socially mature while still at a younger age. Recent studies have found that chimpanzees prefer older mates, as is often the case in other primates. Head shaving is a common phenomenon among many tribes and often is done only by males. Interestingly enough, it is sometimes performed in a manner which exaggerates the natural balding patterns. For example, the South American Yanomano shave the crown of the head, leaving a wreath of hair. Scars from battle are exhibited in this manner. Another South American tribe, the Tchikrin, pluck all facial hair, even in small children. The men, especially, have their hair plucked back from the forehead to the crown or hair whorl area. It was a common practice during the 1800's for Chinese males to shave the forehead well back to the top of the head and then braid the remaining hair in a queue. In the distant past, the gloss of a bare scalp became the badge of leadership and dominance, whether it was the greased plucked head of the Yanamano or the oily, scraped scalp of an Ainu, Jew, Chinese, or Saxon. It is mimicked unconsciously by shiny metal helmets in many cultures. A low hairline has a more juvenile appearence, and is a sign of youth. The evolution of human scalp hair has followed this pattern: first it was an erectile threat crest, then strangely, it began to shift. Balding became the threat ideal, and a full head of soft hair was what we clung to as babies - a symbol of maternal-sexual security and attraction, like a round, warm breast. But recently the evolutionary bent has looped into an even odder twist. The symbols of age and status are in disfavor, even repugnant. Now it is the mature male who mimics the post-puberty vigor of youth that has become our man's ideal. We live in a society which bases most status evaluation on one's potential courting currency; that is the secret behind our reverence for youth. AGA is characterized by higher levels of the potent androgen dihydrotestosterone in the scalp, and increased expression of the androgen receptor. The gene ordering the production of the androgen receptors is polymorphic as it can contain a various number of CAG repeats. In normal people, the CAG repeats can range from 6 to 39. It means that in our gene encoding androgen receptors, the CAG triplets can be repeated 6 times up to 39 times. It seems obvious that each polymorphism will produce a slightly different kind of androgen receptors. Up to 23 repeats, one is considered as having short CAG while above 23 one belongs to the long repeat group. People with short repeats are the most sensitive to the androgenic actions of testosterone, whereas people having the longest repeats are less sensitive to the androgenic actions of testosterone. AGA has been associated with shorter CAG repeats. It is likely that men with the AR varient associated with AGA have more of the enzyme 5a-reductase. 5a-reductase converts testosterone into the more potent androgen dihydrotestosterone.

The quen to the question is, is hydrous existisity a factor in the surplus dam of testosterone in females?

The exactness of the situation is that all females carry testosterone.

So therefore this can be expressed in parallel as female hyenas as having pensies.

So therefore in microcosm, all females who have an excess of testosterone, therefore can adopt (the strong traits).

Furthermore, to cut back to the question, is expressed as /

So in what level of testosterone that is deemed is bad, does have a bearing effect on the set of all females as being bald?

This must be done by survey.. So the redressed question must be expressed, if the follicles of a female who suffers from female pattern baldness is such that dominancy is expressed as in tricho-resession, as trico being hair, then of what percentage of those females is the lack of hair growth related to males who suffer the same problem?

Under the hyduscus true