I discovered this years ago....

Benzene, Lubricants and AIDS

Well for me it was an alarming article because I was working with Phenol, an acidic form of Benzene and this was confirmed for me when I got these symptoms a couple of years later....

They were rapid weight loss, night sweats, severe insomnia, lymph swelling, nausea, shakes and headaches.
This later led to my white blood cells rushing to my skin where thousands of pores filled with pus, my skin fell off, I had lesions, fevers, chills and was later told I could have gone into a coma....

So I found myself doing this interview a few years later.

Benzene, Corticosteroids and AIDS

So I know about the T-cell depletion caused by Benzene but I've found this on a disc lately and as far as I know with a Google search this is not on the net.
So I'll post it if curious.
The main question I'd like to ask is there anyone here who understands the chemistry Lamb is talking about, it's not my field, I have more knowledge of nutrition and how it affects retroviruses and the genome....later.

I'd be very grateful if anyone has the know how to explain him here.

I'm also completely well now, yes

This is me by the way....
Cal Crilly
Star Sludge

Thanks to Physorg for a forum too
CU's


BENZENE HYPOTHESIS OF AIDS - DOCUMENTATION

The biochemistry of AIDS has been studied for nearly a decade, and the fact that some abnormal biochemical processes are occurring is well documented. A computerized literature search of the American Chemical Society database reveals that they have over 6,000 documents that deal with this chemistry. A search of this DIALOG database using the keywords HIV and protease turned up over 600 documents that describe the chemistry of AIDS. Most of these documents contain an x-ray structural analysis of the carbon, nitrogen and hydrogen bonding arrangements, as well as the position of the other elements, compounds and amino acids that are involved.
The problem is in interpreting what these arrangements represent. The HIV Hypothesis has been proposed to explain these unusual phenomena. Accordingly the treatment strategies resulting from the application of the HIV theory utilizes toxins to try to kill the HIV, which they claim is the cause of these conditions. This approach has not been successful in saving even a single human life. Whereas these toxin treatment strategies, having excluded all other alternative procedures, have resulted in the death of over 270,000 people in the United States alone.
I will present here another explanation to account for these phenomena and document my hypothesis by existing facts and known laws of chemistry, biochemistry and physics which the HIV theory has never tried to do. The attributes of HIV, according to the widely accepted theory, defy all known principles of chemistry and biochemistry, and a whole new language and new principles were developed to explain these observed phenomena.
I will present the HIV theory’s explanation in their own unique terms and compare that with my explanation of the phenomena for each of the key points addressed.
As for documentation, I refer to the text, Biochemistry, Fourth Edition, 1995, by Lubert Stryer, published by W. H. Freeman and Co., NY; ISBN: 0-7167-2009-4. There are several figures presented that would be enlightening and beneficial to the understanding of the mechanics of both the HIV and the Benzene Hypothesis. I will include the pages and figure numbers, where appropriate.
To document the Chemical Ionization Potential of Elements
and Compounds, you can refer to Appendixes A and B in my
book, How I Cured Myself of AIDS or any other suitable
reference from the library.


HIV Theory - Point 1
ATCase starts with tyrosine going to the gut where the Escherichia coli convert it to aspartate transcarbamoylase (ATCase). The tyrosine is presented as R-CH{2}-figure [a hexagon shaped 6- carbon ring with alternate double bonds]
 OH. [ Note: {} denotes subscript.]


The very first ASSUMPTION of the HIV theory is that the HIV causes the tyrosine to form biotoxic masses that replicate and eventually take the life of the victim.

Benzene Theory - Counter Point 1
This is also the symbol and arrangement of the hexagon shaped 6-carbon ring with alternate double bonds derived from petrochemical sources called benzene.
The very first ASSUMPTION of the HIV theory is wrong! Tyrosine is a bioactive amino acid. Benzene is a biotoxic compound that has high energy bent carbon-carbon bonds that are much harder to separate than tyrosine.
All of the so-called HIV enzymes, chemical synthesis, toxin treatment strategies and the whole vocabulary of HIV technology is based on the false assumption that tyrosine is the native trimmer found in the body that starts the whole protease synthesis process.
The native trimmer is in reality benzene that has been acquired by prolonged exposure of the individual to benzene toxin laden air, benzyl containing food or drink and/or benzyl containing personal products such as lubricants.

HIV Theory - Point 2
That there is a level of human immunodeficiency virus type 1 (HIV-1) protease activity that is required for HIV-1 particle maturation and infectivity. This is documented in the Journal of Virology, 1995, Vol. 69, Number 5, Pages 2751-8 by Rose, Jason R.; Babe, Lilia M.; Craik, Charles S.; of the Department of Pharmacology, University of California, San Francisco, CA 94143-0446. ISSN: 0022-538X in their article, “Defining the Level of Human Immunodeficiency Virus Type 1 (HIV-1) Protease Activity Required for HIV-1 Particle Maturation and Infectivity.”

Benzene Theory - Counter Point 2
All this level is showing is that there is a critical level of benzene and benzyl compounds that is required to initiate the synthesis of benzyl compounds in the body. There are also many unnatural and unhealthy conditions that must also develop to enable this benzene synthesis. These include greatly diminished levels of niacin related enzymes NAD and/or NADP, many other vitamins and minerals and the lowering of the normal ph of the various systems of the body.


HIV Theory - Point 3
That Escherichia coli convert tyrosine to aspartate transcarbamoylase (ATCase) which is critical for the active form of HIV-1 protease.
This is documented in the article, “Expression and
Purification of Active Form of HIV-1 Protease from E. coli,” by Wan, Min; Loh, Boon Nee, published in Biochem. Mol. Biol.
Int., 1995, Vol. 35, Number 4, Pages 899-912, ISSN:
1039-9712.
It is also documented in the article, “Large-scale Production of HIV-1 Protease from Escherichia coli Using Selective Extraction and Membrane Fractionation,” by Gustafson, Mark E., et. al. which was published in Protein Expression Purif., 1995, Vol. 6, Number 4, Pages 512- 18, ISSN: 1046-5928.

Benzene Theory - Counter Point 3
The USDA have demonstrations using E. coli to process orange peelings and synthesize the hydrocarbon ethanol. If under ideal conditions E. coli can synthesize ethanol, there should be no reason they can not synthesize benzene after the benzene starter, catalysts and other chemical requirements have been met.
The E. coli are critical to the development of the active form of the HIV protease because the benzene and benzyl compounds are synthesized by them in the gut after a critical level of these toxins are reached.

HIV Theory - Point 4
Aspartyl protease is required for replication of HIV. That this replication requires proteolytic cleavage of a polyprotein that contains several key viral proteins. The proteolysis is carried out by a virus-encoded protein that must first separate itself from the polyprotein. Then it hydrolyzes additional peptide bonds in the polyprotein to free the other essential viral proteins.
The fact that there is a sequence of three amino acids, Asp-Thr-Gly (Aspartic Acid, Thyreonine, Glycine) in the HIV-1 protease suggested that it is an aspartyl protease. This is because this sequence of amino acids occurs at the several active sites of the class of enzymes. However, HIV-1 protease is not quite half the size of these other aspartyl proteases and contains only one such amino acid sequence. (Page 229, Figures 9-42, 9-43, 9-44, 9-46.)

Benzene Theory - Counter Point 4
The benzene section frees itself of the attached protein components. Since it carries with it one nitrogen atom, they classify it as protein. Due to the high chemical attraction, the benzene ring fragment separates a carbon and a nitrogen from the protein to complete the ring structure.
Since HIV-1 is not quite half the size of the other aspartyl proteases, it can not truely be called aspartyl protease. This is because it is composed of half of a benzene ring fragment containing three benzene carbons. It would be more appropriate to call it benzyl protease.

HIV Theory - Point 5
The pyrimidine ring is synthesized from carbamoyl phosphate and aspartate. The origin of the atoms in the pyrimidine ring is shown in Figure 29-10, page 745. The C-2 and N-3 come from carbamoyl phosphate, whereas the other atoms of the ring come from aspartate.
If you number the six atom hexagon ring in a clockwise rotation starting at the bottom, or 6 o’clock position, one is a nitrogen from aspartate. Two is a carbon and three is a nitrogen, both from carbamoyl phosphate. Carbons 4, 5 and 6 complete the ring and they are from aspartate. In total the ring has three carbons and one nitrogen from aspartate, and one carbon and one nitrogen from carbamoyl phosphate.

Benzene Theory - Counter Point 5
The pyrimidine ring they refer to is not a normal pyrimidine ring even though it has the same number of atoms of carbon and nitrogen as the bioactive pryrimidine. It is biotoxic since the three carbons from the aspartate are from benzene. The high energy bent bonds of the benzene portion cause this “false pyrimidine” ring to react differently than the normal form. For lack of a better term, I will refer to this grafted ring compound as Benzyl-pyrimidine.

HIV Theory - Point 6
The synthesized pyrimidine forms N-carbamoylaspartate from the aspartate and carbamoyl phosphate. This reaction is catalyzed by aspartate transcarbamoylase (ATCase), which is a regulatory enzyme. This ATC enzyme comes from E. coli and has been studied by many investigators.
The ATCase is feedback inhibited by cytidine triphosphate (CTP). John Gerhart and Arthur Pardee found that the binding of carbamoyl phosphate and aspartate are mutually cooperative. The reaction velocity is dependent upon the substrate concentration. (Figure 10-2.) The higher the concentration of aspartate, the faster the rate of reaction will be. The normal body energy unit, ATP (Adenosine Tri-phosphate) is an activator of aspartate transcarbamoylase.
The enzyme cytidine triphosphate (CTP) is made by E. coli and inhibits aspartate transcarbamoylase.
The ATCase is regulated by CTP and ATP and has great biological significance. The availability of ATP provides the energy for DNA replication and causes the synthesis of the pyrimidine nucleotides. Also that inhibition by the CTP prevents N-carbamoylaspartate and the subsequent compounds in the pathway are not formed when the pyrimidines are abundant.
Benzene Theory - Counter Point 6
The benzyl-pyrimidine (or false pyrimidine) would bind to the carbamoyl phosphate donated carbon and nitrogen the way the aspartate transcarbamoylase supposedly reacts.
However, the benzyl-pyrimidine would accept the electron from the ATP compound which would drive the formation of more ATCase or benzyl-compounds. What this represents is an electrically driven hydrocarbon synthesis process that is catalyzed by the unique chemistry that develops in these locations of the body where this synthesis is occurring.
The CTP does not carry an electron, and thus, when it binds to the bonding site, so that ATP can not bond there to donate the electron, it inhibits the hydrocarbon synthesis process.
Normal pyrimidine would use the ATP energy to further the normal life giving biochemical reactions. However, the benzyl-pyrimidine uses the ATP energy to synthesize more biotoxic hydrocarbons. This accounts for the illogical reactions that are observed with these conditions. The HIV therory has no explanation for how or why these mechanisms exist. The benzene hypothesis puts these conditions in clear perspective and in accordance with the known laws of chemistry, biochemistry and quantum physics.

HIV Theory - Point 7
The regulatory mechanism of ATCase disappears when you treat it with a mercury containing compound such as p-hydroxymercuribenzoate. ATP and CTP have no effect on the ATCase after it has been treated with the mercury containing benzoate. The binding of other substates to ATCase also becomes uncooperative after this treatment. However, the ATCase that has been treated with the mercuribenzoate has the same catalylic activity as the original ATCase. (Figure 10-3, p. 239.)
Studies of this mechanism were carried out by John Gerhart and Howard Schachman. They did ultracentrifugation studies to find that the mercuribenzoate would dissociate ATCase into two kinds of subunits. The sedementation coefficient of the original ATCase was 11.6 S, but the two disassociated subunits, after treatment, were 2.8 S and 5.8 S. These two subunits can be separated by ion-exchange chromatography because they have a markedly different charge. They can also be separated by centrifugation in a sucrose density gradient since they are different in size.
The largest subunit is called a catalytic subunit since it is catalytically active. However, it is unresponsive to ATP and CTP. Since the smaller subunit is devoid of catalytic activity, it is called a regulatory subunit. But it can bind CTP and ATP.


Benzene Theory - Counter Point 7
The three carbon benzene segment would be catalytically active due to the covalent bonds which leave it a strongly charged compound; therefore, it would react with other compounds in its normal state. The chemical ionization potential of benzene is 9.25 (Lamb Appendix . The chemical ionization potential of mercury (Hg) is 10.43. (Lamb Appendix A). The carbons in the benzene have a valence of plus or minus 4 or 2. The valence of the mercury is 2 or 1. Therefore, the mercury in the mercuribenzoate would bond to the benzene section, which would account for why the binding of other substrates would be uncooperative after this treatment. Once the mercury is bonded to the benzene ATCase section, the remaining benzene has the same catalytic activity as the original benzene compound. Of course, the benzene in the treatment is going to react the same as the benzene in the ATCase. Benzene is benzene!
The other compound from the biological source would not catalyze any reactions since it does not normally cause such reations. In other words, this is normal biochemical behavior. Many elements, compounds and amino acids are used in the synthesis of hydrocarbon compounds and hydrocarbon polymers by petrochemical engineers.

HIV Theory - Point 8
Using x-ray analysis, the structure of ATCase and its complex have been solved. William Lipscomb and co-workers developed the three-dimensional structure of ATCase with a 2.6 angstrom resolution. The enzyme contains a large central cavity. Looking down it has a threefold symetrical axis. Each of the three symetrical units are related by 120 degrees of rotation from each other. Each regulatory subunit is on the outside of the catalytic subunit. Each regulatory subunit is attached to the catalytic chain on the inside and has a binding site for CTP on the outer edge. The inner lobe of the regulatory chain contains a zinc ion. The zinc controls the structure by coordinating the sulfur in the four cysteine residues. The three catalylic subunits are all identical and equally spaced at 120 degrees around a central axis.
The diameter of a sphere that would encompass this ATCase structure is about 13 nanometers (130 angstroms) in size. That is roughly twice the size of hemoglobin. (Figure 10-5, p. 240)

Benzene Theory - Counter Point 8
The 120 degree angle of the three symetrical units is exactly the angle of the three alternate double bond faces of a benzene molecule. This is exactly the structure that would form in a benzene hydrocarbon dimmer and benzene polymer synthesis. Several examples of similar structural relationships can be found in polymerization research published in the American Chemical Society Journal. Any hydrocarbon polymer engineer should recognize the chemical reactions and structures that are formed as typical for the hydrocarbon benzene when combined with amino acids and catalysts that are supplied by the body’s chemistry.

Summary

It is the position of the Benzene Hypothesis that:
1.All 29 AIDS defining conditions result directly or indirectly from prolonged exposure to benzene and benzyl compounds in the aeromatic group of hydrocarbons.
2.That this prolonged benzene exposure is due to:
A)The addition of toxic benzyl compounds to food, either intentional as printed on the ingredients or accidental from atmospheric pollution trapped in the food or water as described by Dr. Hulda Clark in her recent book, The Cure for HIV and AIDS.
B)The addition of 1 to 2 percent benzene to all the gasoline supplies of the world as an anti-detonation compound. That this uncombusted benzene is volatolized and emitted from motor vehicles as an invisible toxic cloud that surrounds cities and high traffic areas as presented in my book, How I Cured Myself of AIDS.
3.That these benzene compounds are stored in the human body as a result of prolonged exposure combined with inadequate nutritional intakes and inadequate amounts of sunlight or ultraviolet light for detoxification.
4.That these benzyl compounds react with the biochemistry of the human body and replace similar bioactive compounds with the biotoxic petrochemical benzyl components, which react differently in the body.
5.That the ELISA and Western blot HIV antibody tests are actually detecting antibodies to benzene and benzyl compounds. That the polymerase chain reaction test is actually measuring the benzene polymer chain reaction level. That the viral load test is actually a hydrocarbon load test, which is primarily benzene. That all 29 AIDS defining conditions are in reality symptoms of hydrocarbon poisoning, either directly or indirectly.
6.That once these benzyl hydrocarbons reach a critical mass in the body, they undertake a catalyzed electrically driven process that allows the benzyl compounds to self-replicate and produce more of these unnatural benzyl hydrocarbons and benzyl polymers. The minerals and amino acids of the body are the same as those commonly used by hydrocarbon polymer engineers and the electrons generated by the body’s energy storage (ATP) and nervous system electrons are used to perpetuate the hydrocarbon replicating process.
7.The HIV hypothesis has no answer for how to reverse and cure these conditions once they develop.
The Benzene Hypothesis clearly identifies the treatments and therapies that can reverse and cure these conditions.
A) Preventing or minimizing continued exposure to benzene and/or benzyl compounds is the first step to effective treatment. If you live or travel in a major city or along a major traffic route, you should consider an air filtration system for your home, car and/or workplace that is capable of removing benzene and other hydrocarbon toxins from your breathing environment. And minimize your benzene exposure in your food, water and personal products.
B)Take ultraviolet light whole body radiation therapy to break down the benzene toxins in your body. UV-A or UV-B both will photo-disassociate benzene so that it is nontoxic to the body.
Prolonged therapy with daily “tanning treatments” will greatly facilitate the benzene detoxification process.
C)Take therapeutic levels of fresh lemons, vitamin C, B-3 (niacin), B-6, B-12, B-complex, sulfur containing amino acids, vitamins, minerals and nutritional supplements as are identified in the Protocol to restore the normal biochemistry and eliminate benzene from your body.
D)When the benzene is all detoxified and eliminated, the benzene antibody level will drop and the ELISA and Western Blot tests will show that you have been seroconverted to antibody negative. With continued therapy you will be cured of these Acquired Benzene Conditions.
Respectfully, William A. Lamb, Ph.D. AIDS Treatment Consultant & Alternative Therapy Provider Behr & Lamb, Inc.;
Rt. 1- Box 298; Jerico Springs, Missouri; 64756-9305; USA Ph
417-398-2448; Fax 417-398-2488; E-mail aids_cure at delphi.com
Copyright Dec. 1995 by William A. Lamb, Ph.D. Permission for non-commercial use of this material is hereby granted so long as the copyright and my contact information remains intact.

You are a dangerous idiot preaching lunacy.

http://aids.about.com/cs/opposingviews/a/connection.htm
http://aids.about.com/cs/aidsfactsheets/a/hivlife.htm
http://www.sfaf.org/aids101/hiv_disease.html

You know no biochemistry, virology, immunology or medicine and you write this wish list?

Abuse will get you nowhere...you've obviously not even looked or thought about it.
So does anyone understand Lamb's article.
Benzene is something real that depletes T-cells and people who believe HIV causes AIDS have done absolutely nothing to inform people with AIDS that this could cause their AIDS.

I finally removed the Benzene from my body with Niacin or B3.
Is it a co-incidence?

"A pentad of findings consistent with niacin depletion have been described in patients with AIDS. There are also clinical and laboratory data to support the potential benefit of niacin in HIV infection. In this paper, it is hypothesized that HIV infection induces niacin depletion, and that therapeutic niacin will act as an AIDS preventive factor. While viral inhibition is incontrovertibly the primary 'AIDS preventive factor', costly antiretroviral medications are simply out of reach for the majority of the world's HIV-infected people. Along with antiviral research, investigation must go forward to look at strategies to overcome the massive metabolic disruption caused by the production of approximately one billion virus particles per day. Niacin, the same B complex vitamin found in the early part of this century to be the 'pellagra preventive factor', is proposed here as a secondary AIDS preventive factor' in HIV-infected persons."
Niacin as a potential AIDS preventive factor

Think a bit harder

Benzene is bad for the immune system, we all breath it from car exhausts, drug users stick Benzene, residues into their arms, the highest rates of AIDS in South Africa are next to the petrol refineries.
So take it seriously.

1.6.4 Immunotoxicity Benzene depresses the proliferative ability of B- and T-cell lymphocytes. Host resistance to infection in several laboratory species has been reduced by exposure to benzene.

1.7 Effects on humans It is known that benzene produces a number of adverse health effects. The most frequently reported health effect of benzene is bone marrow depression leading to aplastic anaemia. At high levels of exposure a high incidence of these diseases is probable.
INTERNATIONAL PROGRAMME ON CHEMICAL SAFETY. BENZENE

Now we’ve also been using a drug called AZT on gays, women and children for over 20 years.
It’s bad for the immune system, there has never been any evidence that it saves a single life but HIV dogmatists continue to shove it down people’s throats.

“At concentrations similar to those reported in clinical trials, 3'-azido-3'-deoxythymidine was found to suppress the mitogen and antigen induced proliferation of T-cells from seronegative subjects. In contrast, B-cell proliferation and the distribution of membrane markers appeared to be unaffected by the drug. Furthermore, 3'-azido-3'-deoxythymidine did not alter the i vivo immunoglobulin secretion capacity in autologous or allogeneic cultures of lymphocytes from seropositive subjects.
In human immunodeficiency virus-infected individuals the number of unactivated, circulating B-cells is significantly reduced. The present results indicate that the functional capacity of these 'remaining' cells may be seriously impaired during antiviral therapy with 3'-azido-3'-deoxythymidine.”
Effects of 3'-azido-3'-deoxythymidine (AZT) on short-term cultured human peripheral blood lymphocytes 1988

“We studied the effects of azidothymidine (AZT) on rat bone marrow heme metabolism and colony growth as determined by assays of granulocyte-macrophage (CFU-GM), erythroid (CFU-E), burst-forming erythroid (BFU-E), and -aminolevulinic acid synthase (ALAS), the first enzyme in the heme pathway. In all cases, AZT (1-0.01 M) was found to be toxic to bone marrow colony growth.”
Role of heme metabolism in AZT-induced bone marrow toxicity 1989

This study is definitive…and involved 22,217 HIV positive people in Europe and North America, including many from sub-Saharan Africa, who had never taken ARVs before, and about 75% had no AIDS symptoms at the start of medication.

“Interpretation: Virological response after starting HAART improved over calendar years, but such improvement has not translated into a decrease in mortality.”

“p 427, “The major findings are that, despite improved initial HIV virological control (percentage there were no significant improvements in early immunological response as measured by CD4-lymphocyte count, no reduction in all-cause mortality, and a significant increase in combined AIDS/AIDS related death risk in more recent years.”

“importantly, the recent increase in AIDS risk seemed largly because of increase tuberculosis incidence.”

“We noted that the median time to the first AIDS event after starting HAART decreased over time”.
May MT et al. HIV treatment response and prognosis in Europe and North America in the first decade of highly active antiretroviral therapy: a collaborative analysis.

Or read The Lancet’s page 451 of Vol 368, August 5, 2006.

So what is the result of all this?
We are still at square one, until AIDS drugs are taken off the market and the possibility that Benzene and poppers caused the initial AIDS epidemic we will never know if there ever was a single cause of AIDS.

Robert Gallo also put the HIV antibody test onto the market when only a third of his patients were positive to his test, that still means two thirds of the AIDS cases were caused by something else.

I haven’t even started yet so remember anything you say will be seen for posterity…
And I'm going to make sure you all have fun thinking about it....so please don't get upset.
I'll be doing the post on retroviruses in cancers and other diseases over the next week
CU’s soon.

Obviously, this topic is controversial, and no matter how pure one's intentions are, the shallow thinkers will automatically accuse you of being "anti-gay" if you propose a theory that runs contrary to the popular dogma.

I'll go on record (pointlessly, just watch) as saying that I have nothing against gays nor am I a pro-conspiracist in any way.

That said, I do recall that when AIDS first hit the news, French doctors pointed out the similarities between AIDS and "graft-vs.-host" ("GVH")syndrome, where the body's immune system gets confused about its own identity and attacks its own bone marrow along with various other parts of the body.

Why would there be similarities between AIDS and GVH?

Sperm cells are even stupider than guys are during sex. Sperm cells are looking for an egg to impregnate, dumping their genetic load into the egg (much like a retrovirus does). The least differentiated cells in the human body, the ones closest to human egg cells, are the stem cells in the bone marrow responsible for making our immune system's components. During gay sex, it's rather commonplace for sperm cells to gain access to their partner's bloodstream. If a sperm cell remains viable in the bloodstream long enough to reach his bone marrow, it's conceivable (not proven, but within the realm of possibility) that the sperm cell could mistake a stem cell for an ovum and dump its genetic load into the cell, confusing that stem cell's identity. That stem cell would thereafter create immune cells that would attack other (original) marrow stem cells as "invaders", and you'd get the classic set symptoms of Graft-versus-Host disease (which, IIRC, includes Kaposi's Sarcoma).

If this is true, then there would be no real link between HIV-1 and AIDS. Oddly, there are AIDS patients who aren't HIV-positive, and a lot of HIV-positive patients who don't develop AIDS. A lot of golfers develop skin cancer of the scalp, but there's nothing intrinsic to the game of golf that can be considered *causative* of skin cancer. Golfers spend more time outside than most non-golfers, and the sun causes skin cancer. Gay sex exposes gay men to various diseases, not just HIV, but Hepatitis-A and various STDs, it's possible that HIV is just another STD that's more prevalent in the gay community.

You have to understand the problem before you can find the solution, and dogma often gets in the way of understanding. It's refreshing to know that there are some educated people out there willing to think "outside the box."

Incidentally, I have no strong opinions one way or the other whether AIDS is caused by HIV or by sperm cells entering the bloodstream, and no vested interest in either explanation other than hoping that someday people will discover the cause and an effective cure for AIDS.

If HIV-1 does truly cause AIDS, then I'd recommend pursuing this line of research:

http://www.jnanobiotechnology.com/content/3/1/6

"Interaction of silver nanoparticles with HIV-1
In this work, we demonstrate that silver nanoparticles undergo a size-dependent interaction with HIV-1, with nanoparticles exclusively in the range of 1–10 nm attached to the virus. The regular spatial arrangement of the attached nanoparticles, the center-to-center distance between nanoparticles, and the fact that the exposed sulfur-bearing residues of the glycoprotein knobs would be attractive sites for nanoparticle interaction suggest that silver nanoparticles interact with the HIV-1 virus via preferential binding to the gp120 glycoprotein knobs. Due to this interaction, silver nanoparticles inhibit the virus from binding to host cells, as demonstrated in vitro."

Silver nanoparticles are, for the most part, non-toxic, and colloidal silver is relatively cheap and effective against a wide range of pathogens. The biggest problem with it seems to be "argyria", which, if you're purchasing the recommended concentrations of 10ppm (parts-per-million), you'd need to ingest about 3 tons of the stuff before turning blue. Taken at a reasonable dose, it'd take about 500 years to ingest enough silver for a person to turn blueish. I've personally taken a couple gallons of the stuff over the past couple of years, and it's worked on virtually everything I've tried, including an abscessed tooth, a chronic (over ten years) sinus infection, bronchitis, and it's cured every cold I've started coming down within a day for the past two years, at least.

And for the record, I've never sold a drop of colloidal silver in my life, but I've given it away to all my friends who routinely are surprised that it actually works as I've said it does. One thing, though, it can't tell the difference between acyclovir and a virus, so if you have herpes and are taking acyclovir to suppress outbreaks, taking colloidal silver will almost certainly cause a herpes outbreak, so use your own judgment as to whether it's worth it to you. (I discovered this when I gave colloidal silver to a friend of mine who has Hep-C, and she also had herpes but hadn't mentioned it, and the friendship suffered as a consequence.)

Ugh.

Just because Benzene can cause immunological deficienices doesn't make Benzene the sole cause of AIDS.

First description of AIDS as a well-defined disease: 1981
First industrial production of (benzene, bicarburet of hydrogen, benzin): 1849.

An incomplete list of clinical descriptions of benzene poisoning between these dates:

• Aksoy M, Dincol K, Akgun T, Erdem S & Dincol G (1971) Haematological effects of chronic benzene poisoning in 217 workers. Br J Ind Med, 28: 296-302.
• Aksoy M, Dincol K, Erdem S, Akgun T & Dincol G (1972) Details of blood changes in 32 patients with pancytopenia associated with long-term exposure to benzene. Br J Ind Med, 29: 56-64.
• Aksoy M & Erdem S (1978) Followup study on the mortality and the development of leukemia in 44 pancytopenic patients with chronic exposure to benzene. Blood, 52: 285-292.
• Aksoy M, Erdem S & Dincol G (1974) Leukemia in shoe-workers exposed chronically to benzene. Blood, 44: 837-841.
• Aksoy M, Erdem S & Dincol G (1976) Types of leukemia in chronic benzene poisoning. A study in thirty-four patients. Acta Haematol, 55: 65-72.
• Andrews LS, Lee EW, Witmer CM, Kocsis JJ & Snyder R (1976) Effects of toluene on the metabolism, disposition and hemopoietic toxicity of [3H]benzene. Biochem Pharmacol, 26: 293-300.
• Berlin M, Holm S, Knutsson P & Tunek A (1979) Biological threshold limits for benzene based on pharmacokinetics of inhaled benzene in man. Arch Toxicol, 2S: 303-310.
• Brief RS, Lynch J, Bernath T & Scala RA (1980) Benzene in the workplace. Am Ind Hyg Assoc J, 41: 616-623.
• Cornish HH & Ryan RC (1965) Metabolism of benzene in nonfasted, fasted, and aryl-hydroxylase inhibited rats. Toxicol Appl Pharmacol, 7: 767-771.
• Cronin HJ (1924) Benzol poisoning in the rubber industry. Boston Medical and Surgical Journal, 191: 1164-1166.
• Dowty BJ, Laseter JL, Storer J (1976) The transplacental migration and accumulation in blood of volatile organic constituents. Pediatr Res, 10: 696-701.
• Drew RT & Fouts JR (1974) The lack of effects of pretreatment with phenobarbital and chlorpromazine on the acute toxicity of benzene in rats. Toxicol Appl Pharmacol, 27: 183-193.
• Flury F (1928) Moderne gewerbliche vergiftungen. IIa: Moderne gewerbliche vergiftungen in pharmakologisch-toxikologisher hinsicht. Naunyn Schmiedebergs Arch Exp Pathol Pharmakol, 138:65-82 (in German).
• Forni AM, Cappellini A, Pacifico E, Vigliani EC (1971) Chromosome changes and their evolution in subjects with past exposure to benzene. Arch Environ Health, 23: 385-391.
• Funes-Cravioto F, Zapata-Gayon C, Kolmodin-Hedman B, Lambert B, Lindsten J, Nirberg E, Nordenskjold M, Olin R, Swensson A (1977) Chromosome aberrations and sister-chromatid exchange in workers in chemical laboratories and a rotoprinting factory and in children of women laboratory workers. Lancet, 2: 322-325.
• Gerarde HW (1960) Toxicity and Biochemistry of Aromatic Hydrocarbons. Elsevier, New York.
• Goldstein BD (1977) Hematotoxicity in humans. J Toxicol Environ Health, 2S: 69-105.
• Green JD, Leong BKJ, Laskin S (1978) Inhaled benzene fetotoxicity in rats. Toxicol Appl Pharmacol, 46: 9-18.
• Greenburg L (1926) Benzol poisoning as an industrial hazard. Public Health Rep, 41: 1357-1375.
• Haley TJ (1977) Evaluation of the health effects of benzene inhalation. Clin Toxicol, 11: 531-548.
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So the clinical picture of acute and chronic benzene poisoning was well-described well-prior to 1981. It is unreasonable that any M.D. would confuse the two, or that this alleged misclassification would have gone unnoticed by those M.D.s who specialize in benzene poisoning.

Unless you are up-to-date on mechanisms of immunodeficiency in AIDS and benzene poisoning you are an idiot to think that you have a match when the actual mechanism of HIV/AIDS is it works the immune system to death. (wcelliott, that's why it looks like GVH, because the immune system's memory of safe and self is destroyed and the resulting immnune storm worked the immune system to death.)As you are advocating "nutritional" cures without demonstration of efficacy, you are preaching lunacy. As you are preaching to the uninformed masses instead of the relevant health care community, you are dangerous -- inserting noise into a life-and-death struggle for useful information.

Q.E.D. You, Star Sludge, are a dangerous idiot preaching lunacy.
References copied from http://www.inchem.org/documents/pims/chemical/pim063.htm

Solvents and the Kaposi's Question?

This next bit on Kaposi's should help.

Kaposi’s Sarcoma is a cancer. It is also the star disease of HIV/AIDS Inc. And has been the nasty one that knocked over a lot of people. It isn’t a ‘gay disease’ though, none of the AIDS diseases are.
Kaposi’s occurs in other population, I noticed Harare has a high prevalence and historically there’s Sardinia.
Direct correlation between human herpesvirus-8 seroprevalence and classic Kaposi’s sarcoma incidence in Northern Sardinia.
http://www.ncbi.nlm.nih.gov/entrez/query.f...6&dopt=Abstract

If Robert Gallo could just hold a press conference and declare HIV to be the ‘probable cause’ of AIDS I’ll go one further and answer the Kaposi’s question once and for all.

The Scientists who blame HHV8 Herpes for Kaposi’s Sarcoma are right.
Peter Duesberg blaming the Nitrite solvents that people inhale for increased sexual pleasure is right.
Dr. Bayati who blames Corticosteroids for switching off the immune system is right.
All the Cancer and AIDS researchers who see HIV and HERV-K retroviruses are looking at something real but that’s a side effect of the Collagen Matrix collapsing. Harold Foster and his use of Selenium to help AIDS sufferers in Africa is right.
Even Hulda Clarke blaming Benzene and other solvents for allowing parasites to flourish and wreck the Thymus gland is right. Parasites will release growth hormones and encourage Cancer, what Hulda missed was that the solvents dissolve Collagen and Herpes viruses will have a party with those conditions.
The parasites are very likely to be a factor in Prostate Cancer so her cures with anti-parasite herbs often work with Prostate but the Herpes factor is a big one she missed.
The lipid bilayer or our skin and why it aligns itself in a certain way was discovered in 1917.
In 1917,Irving Langmuir discovered that phosphilipids dissolved in benzene would form a film on water when the Benzene evaporated. In other words he discovered that when skin dissolves it realigns itself after the solvent that dissolved it has gone. He found our fatty skin has attractive side to water and one that also repels water. Here’s some history of membranes if curious.
http://www1.umn.edu/ships/9-2/membrane.htm

I discovered this while I was doing first year university chemistry part-time and working in a factory with Phenol, an acidic Benzene molecule, it helped explain the nosebleeds I was getting.
Benzene, toluene and acetone—frequently used as solvents of skin carcinogens—have different effects on the skin collagen of mice: Benzene and toluene lower the collagen content significantly whereas acetone does not.
[The effect of various solvents (benzene, toluene, acetone) used with carcinogens on the collagen content of the mouse dorsal skin]
http://www.ncbi.nlm.nih.gov/entrez/query.f...0&dopt=Abstract

So Hulda’s paranoia about Benzene causing AIDS and Cancer has some hefty reasons behind it.
Solvents are dangerous and inhalants are solvents.
Here in Queensland, Australia the effects of two decades of drug war have left the rich school kids dealing pot in schools and the poor white and aboriginal kids sniffing a whole array of solvents.
I’m generalising a little, the inhalant epidemic crosses all the social groups and ages.
I can get on a train and someone can have a coke bottle filled with glue up their T-shirt. The same happens in town waiting for a bus, homeless casually take a sniff or two in conversation with friends who prefer to stick to alcohol or their ciggies but it’s happening right in front of us.
I had chronic glue exposure for 3 years and it doesn’t thrill me what it did to me.
I’ve never been in so much pain, my whole endocrine system seemed to go into hyperdrive and my skin really felt like a chronic chickenpox attack was in full swing.
Kaposi’s and Non Hodgkin’s Lymphoma are the two killers in Western AIDS.
Here is some more about incidence.

Causes, incidence, and risk factors:
Before the AIDS epidemic, Kaposi’s sarcoma was seen primarily in elderly Italian and Jewish men and developed slowly in these people. In AIDS patients, it can develop aggressively and often involves the skin, lungs, gastrointestinal tract and other organs.
Kaposi’s sarcoma
http://www.healthcentral.com/mhc/top/000661.cfm

"The prevalence of KS and lymphoma, lymphadenitis, tuberculosis in Africa is similar to the male homosexuals AIDS patients in US and Europe and even higher (Al-Bayati, 1999). However, AIDS in Africa occurs almost equally in males and females because starvation affects both sexes equally. Sibanda and Stanczuk, (1993) reviewed all lymph node histopathology reports of lymph node biopsy submitted to the Histopathology unit in Harare, Zimbabwe in the period of January 1988 to June 1990. The commonest diseases in the 2,194 lymph node specimens were: non specific hyperplasia (33%), tuberculous lymphadenitis (27%); metastases (12%), Kaposi’s sarcoma (9%); and lymphomas (7%). Kaposi’s sarcoma (KS) involving the lymph nodes was reported in 176 (9%) of the lymph nodes. In children, the prevalence of KS was higher in children under 5 years than in 6-15 year bracket. Approximately two thirds (65%) of all patients with KS were aged between 20 and 40 years"
Causes And Pathogenesis Of AIDS In Africa
http://www.ourcivilisation.com/aids/not/africa.htm

Burkitt’s Lymphoma occurs in Africa and the culprit is often the Glandular Fever Herpes virus, Epstein Barr.
What is Burkitt's lymphoma?
Later research showed that B-lymphocytes in these children had been infected with the Epstein-Barr virus (or E-B virus). Epstein-Barr virus infections are known as glandular fever or infectious mononucleosis: they are common and usually cause no problems, but in central Africa many of the children had chronic malaria infections, which reduced their resistance to the virus. In some cases this allowed the virus to change the infected B-lymphocytes into cancerous cells, leading to the development of the lymphoma. This is known as classical African or endemic Burkitt's lymphoma.
In one type of non-Hodgkin's lymphoma the tumour cells have very similar appearances under the microscope to those of classical African or endemic Burkitt's lymphoma. This rare condition is still called Burkitt's lymphoma but is known as the non-African or sporadic type.
It seems that in this condition, once again, the Epstein-Barr virus infection can develop because the patient has reduced immunity. The Epstein-Barr virus is able to survive and transforms the normal B-lymphocytes into cancerous cells. However, in the Burkitt's lymphoma seen in the UK, the way normal B-lymphocytes change to cancer cells is less clear and not all cases occur in people who have been in contact with the Epstein-Barr virus.
http://www.cancerbacup.org.uk/Cancertype/L...Burkitts#356356

Epstein Barr will deplete Lysine and put pressure on Collagen formation.
The causes are the same in the West or Africa, it’s all about destruction of the Collagen Matrix.
Cities in Africa have shocking pollution and therefore exposure to solvents, same in the West but add inhalants to the mix and those are the people who often go down first.
Starvation in the Third World creates the same effect, people in the unpolluted countryside don’t have enough nutrients to repair their Collagen.
Herpes viruses come out when a Vitamin C and Lysine deficiency occurs and wreck the Collagen in the long run. Cancer and TB both release Metalloproteinase a Collagen destroying enzyme, the correct remedy for all these killers is to build up the Collagen Matrix with the nutrients that are its building blocks.
Here are the warnings that are not getting through to sniffers. From a site for drug fans.
The key points to instil to kids are:
1. Inhalants are different from other drugs.
2. Inhalants rank number 4 in popularity for use
3. Inhalants cause the most body damage when compared to all the other drugs.
4. Inhalants may kill the first time when used.
5. There are over 1400 inhalant products that kids can use to get high.
http://www.erowid.org/chemicals/inhalants/...nts_info4.shtml

Amyl and butyl nitrites have been associated with Kaposi’s sarcoma (KS), the most common cancer reported among AIDS patients. Early studies of KS showed that many people with KS had used volatile nitrites. Researchers are continuing to explore the hypothesis of nitrites as a factor contributing to the development of KS in HIV-infected people.
http://www.focusas.com/Inhalants.html

The following diseases are those officially recognized by VA as related to herbicide exposure.
And included in that list is...
Non-Hodgkin’s Lymphoma, including any diagnosis of a lymphoma [except Hodgkin’s lymphoma], mycosis fungoides, and old terms such as lymphosarcoma, reticulum cell sarcoma and Kaposi’s sarcoma.
V.V.A.’s Guide on AGENT ORANGE
http://www.vba.va.gov/bln/21/benefits/herbicide/AOno3.htm

My point here is that the all people not just AIDS sufferers get Lymphomas and Kaposi’s from Chemical exposure.

I put this next comment in to highlight that AIDS and Cancer patients really die from the same things.

‘Lymphomas frequently develop in HIV positive people. It is expected that the incidence of HIV-related lymphoma will increase as people with AIDS live longer. It is thought that these lymphomas emerge in patients where underlying immunosuppression is coupled with chronic overstimulation of the immune system.’ Note the line ‘chronic overstimulation of the immune system’, this is what chemicals like Benzene do, they push the T-cells to the limit, they fit into endocrine receptors, they burn you out.
Non-Hodgkin’s lymphomas
http://www.geocities.com/~vera_b/origin.html

This also demonstrates where Dr. Mohammed Al Bayati is right, chronic use of coke and amyl nitrate in the gays with GRIDS was causing inflammation of their lungs. Followed with overuse of Corticosteroids that switched off the immune response, another recent fashion, the gays ended up with pneumonia and our friends at the Epidemic Intelligence Service found them and invented an epidemic.
Now I’m often accused of denying the existence of a HIV/AIDS epidemic with such dangerous ideas that call into question HIV theory. I’m saying quite the opposite, because everyone is so obsessed with HIV being the cause of everything we are ignoring the real problems.

This is the heavy stuff about Nitrites you should know about.
Confusion Between Nitrites & Nitrates
http://www.erowid.org/chemicals/inhalants/...nts_info5.shtml

"Since 1983, when he was working at the Centers for Disease Control in Atlanta (CDC), and began analyzing the early data on AIDS, he has been intrigued by the possible role of a widely abused drug called poppers. A nitrite­based inhalant, it just may be a missing key to the endless medical puzzle called AIDS. In particular, Haverkos believes that the drug may be the mysterious cause of Kaposi’s sarcoma (KS), the rare form of cancer that, at the outset of the epidemic, almost defined AIDS. ‘It’s clear that HIV alone can’t explain Kaposi’s,’ he said. ‘There has to be something else’ (Haverkos, 1994).
Haverkos’s career with the Public Health Service was launched just as AIDS was discovered. A Notre Dame graduate, he attended the Medical College of Ohio in Toledo, and did his intern residency at Akron City Hospital. Then, in July, 1981, he joined the CDC in Atlanta. Something new and strange was happening in the homosexual communities on both the East and West Coasts. Young homosexuals, apparently in good health, were coming down with previously rare diseases. One month earlier, five case of Pneumocystis carinii pneumonia had been reported by Dr. Michael Gottlieb in Los Angeles (CDC, 1981a).
‘The patients did not know each other and had no known common contacts or knowledge of sexual partners who had had similar diseases’, Dr. Gottlieb reported. ‘The 5 did not have comparable histories of sexually transmitted disease... Two of the 5 reported having frequent homosexual contacts with various partners. All 5 reported using inhalant drugs...’.
AIDS AND POPPERS
http://www.virusmyth.net/aids/data/tbpoppers.htm

Butyl & Beyond: Pursuing Hex-Tasy
When amyl passed into prescription-only status, a small swarm of little-known chemical cousins crept out of the closet and into the noses and lungs of a new generation of users.
The most popular early stand-in was butyl nitrite, a chemical that differs only slightly from amyl, but packs plenty of the same punch.
Sold as a “room odorizer” or “liquid incense,” to sidestep the U.S. Food & Drug Administration’s regulatory authority, butyl was hawked under such trade names as “Locker Room” and “Jac-Aroma” — which successfully conveyed the awful smell of the chemical: a scent hovering somewhere between month-old mildew and sweaty workout gear.
That didn’t stop the curious, though, from trying them and may even have added to butyl’s cachet in the ‘80s, as use quickly spread from gay bars to dance clubs to the general public.
Still, what happened to amyl eventually happened to butyl, too, as the U.S. Consumer Products Safety Commission stepped in where the FDA couldn’t to ban the chemical in 1988.
That only made the problem morph into something else. New act-alike chemicals appeared in almost-Biblical fashion (Amyl begat Butyl which begat Isobutyl which begat Isoamyl which begat Isopropyl) as each in turn was removed from the market by federal agencies.
Today, the newest nitrite is cyclohexyl nitrite, commonly sold as a “head cleaner” for VCR’s, in a new effort to bypass controls.
Cyclohexyl Nitrites 2000
The nitrites’ most recent incarnation is cyclohexyl nitrate, commonly sold in head shops and adult book stores as a “head cleaner” for VCR’s.
Chemically, cyclohexyl is similar to its predecessors, amyl and butyl nitrite, with an industrial-strength odor that probably helps keep overuse down. Packaging is similar, too, right down to the warning label on the bottle:
Caution: Flammable, harmful if swallowed, skin and eye irritant. If swallowed, drink two glasses milk or water, induce vomiting, call physician. For eye contact, flush with water. Avoid prolonged inhalation in confined areas. Keep out of reach of children.
Ironically, the warning is printed on a plastic sleeve that peels away when the bottle is opened.
Blood Cell Damage. Nitrites damage red blood cells and may cause an often-fatal anemia in which blood can no longer transport oxygen. This type of poisoning happens most often to users who swallow (rather than sniff) the chemical and requires immediate medical treatment.
From ‘Nitrous Oxide & Nitrite Inhalants: Funny Facts’...not that funny really.
http://www.doitnow.org/pages/142.html

Acute blood toxicity of the abused inhalant, cyclohexyl nitrite.
http://www.ncbi.nlm.nih.gov/entrez/query.f...9&dopt=Abstract